Skip Nav Destination
Close Modal
Update search
Filter
- Title
- Author
- Author Affiliations
- Full Text
- Abstract
- Keyword
- DOI
- ISBN
- EISBN
- ISSN
- EISSN
- Issue
- Volume
- References
Filter
- Title
- Author
- Author Affiliations
- Full Text
- Abstract
- Keyword
- DOI
- ISBN
- EISBN
- ISSN
- EISSN
- Issue
- Volume
- References
Filter
- Title
- Author
- Author Affiliations
- Full Text
- Abstract
- Keyword
- DOI
- ISBN
- EISBN
- ISSN
- EISSN
- Issue
- Volume
- References
Filter
- Title
- Author
- Author Affiliations
- Full Text
- Abstract
- Keyword
- DOI
- ISBN
- EISBN
- ISSN
- EISSN
- Issue
- Volume
- References
Filter
- Title
- Author
- Author Affiliations
- Full Text
- Abstract
- Keyword
- DOI
- ISBN
- EISBN
- ISSN
- EISSN
- Issue
- Volume
- References
Filter
- Title
- Author
- Author Affiliations
- Full Text
- Abstract
- Keyword
- DOI
- ISBN
- EISBN
- ISSN
- EISSN
- Issue
- Volume
- References
NARROW
Format
Journal
Article Type
TOC Section
Date
Availability
1-2 of 2
Keywords: CaMK
Close
Follow your search
Access your saved searches in your account
Would you like to receive an alert when new items match your search?
Sort by
Journal Articles
Eun-Ju Chang, Jeongim Ha, Hao Huang, Hyung Joon Kim, Jung Hoon Woo, Youngkyun Lee, Zang Hee Lee, Ju Han Kim, Hong-Hee Kim
Journal:
Journal of Cell Science
J Cell Sci (2008) 121 (15): 2555–2564.
Published: 1 August 2008
... of TRAP + cells to TRAP – cells. The conversion to TRAP – cells occurred with concomitant return to the state with higher expression of macrophage antigens, and greater activity of phagocytosis and dendritic-differentiation potential. JNK inhibition at the pOC stage reduced NFATc1 and CaMK levels...
Journal Articles
Katrin Boels, Günter Glassmeier, Doris Herrmann, I. Björn Riedel, Wolfgang Hampe, Itaru Kojima, Jürgen R. Schwarz, H. Chica Schaller
Journal:
Journal of Cell Science
J Cell Sci (2001) 114 (20): 3599–3606.
Published: 15 October 2001
... second receptor component (Rec) activates a pertussis-toxin sensitive heterotrimeric G protein (1), which interacts via PI3-K (2) with surface GRC (3) to induce Ca 2+ influx (4). The increase in Ca 2+ leads to activation of CaMK (5), which possibly together with PI3-K (?), triggers shuttling of more...
