There was an error published in Development 142, 2194-2202.
On p. 2200, Ng et al. (2012) was incorrectly cited in place of Liew et al. (2014). The corrected text and reference appear below. The authors apologise to readers for this mistake.
IFT27 was recently shown to play a crucial role in facilitating ciliary exit of the BBSome (Eguether et al., 2014; Liew et al., 2014), and Ift27-deficient mouse embryonic fibroblasts are unable to maintain low levels of SMO in the cilia when the Hh pathway is inactive (Eguether et al., 2014).
References
Liew
, G. M.
, Ye
, F.
, Nager
, A. R.
, Murphy
, J. P.
, Lee
, J. S.
, Aguiar
, M.
, Breslow
, D. K.
, Gygi
, S. P
. and Nachury
, M. V
. (2014
). The intraflagellar transport protein IFT27 promotes BBSome exit from cilia through the GTPase ARL6/BBS3
. Dev. Cell
31
, 265
-278
.© 2015. Published by The Company of Biologists Ltd
2015
